| 1. | The defining morphological feature of chemical synapses is the vesicle cluster in the presynaptic nerve terminal
突触小泡即刻可释放池的结构组织化学突触的定义形态学特征是突触前神经终端小泡群。 |
| 2. | A major challenge in studying the synapse is how to tell what observed effects are due to presynaptic factors and which to postsynaptic ones
研究突触最大的困难在于如何区辨所观察到的反应是来自突触前或突触后因子所导致。 |
| 3. | Tiny membranous bubbles called vesicles erupt from each signal - sending , or presynaptic , neuron , releasing serotonin into the synapse
位于讯息发送端的突触前神经元末梢，带有微小的膜状囊泡包裹著血清张力素，可将其释放到突触中。 |
| 4. | The readily releasable vesicles were not clustered close to the presynaptic membrane but instead were dispersed almost randomly throughout the vesicle cluster
结果发现即刻可释放小泡并不是簇集于突触前膜而是随机分散于小泡群中。 |
| 5. | Its neuroprotective effect may be due , at least in part , to interact with presynaptic binding sites and inhibit glutamate and other transmitters release
因此， ginkgolideb的神经保护作用很可能或至少部分是由于它可以与突触前膜的paf受体结合而抑制谷氨酸等神经递质的释放。 |
| 6. | A large proportion of the lab ' s efforts have gone towards finding a technique to directly control the behavior of one side of the synapse ? the presynaptic one ? by replacing it with an artificial terminal under our control
本实验室主要致力于研发新技术，藉由替换一个我们控制之下的人工电极来直接控制突触单一侧(突触前)的行为。 |
| 7. | 6 . at mf - ca3 synapses , both the facilitation ration of ppf and the depression ration of ppd decreased to some degree during the expression of ld - ltp . the result suggested that the expression of ld - ltp at the mf - ca3 synapses may be presynaptic
训练对照组动物随着行为的习得， mf一ca3通路出现了习得性ltp ，且不管是ppd或ppf均在mf一ca3习得性ltp形成后出现改变，这提示mf一ca3习得性ltp形成后的表达与突触前机制有关。 |
| 8. | 7 . whether the dp was induced by the treatment on dneb of 1hz or 5hz delivered 5 min after daily trainings , both ppd and ppf changed during the learning behavior , which suggested that presynaptic mechanisms may be involves in the dp of learning - dependent long - term potentiation
训练后smin不管是使用ihz还是shz刺激dneb产生的dp中，观察到ppd或ppf均在动物训练阶段期间出现改变，这提示习得性ltp的dp可能也与突触前机制有关。 |
| 9. | Due to the toxin ' s pharmacological effect of inhibiting acetylcholine release from presynaptic neuromuscular junctions , botulinum a toxin has been used for the treatment of spastic urethral sphincter as well as detrusor overactivity associated with both neurogenic and non - neurogenic etiologies
基于它在药理学上可以抑制神经肌肉接合前之乙醯胆硷之释放，肉毒杆菌毒素也被应用于神经性因或非神经性因之尿道外括约肌痉挛及逼尿肌活性过强。 |
| 10. | When a neuron fires , sending an electrical signal down its axon to its tips ( presynaptic terminals ) , neurotransmitters released from vesicles cross a tiny intercellular space ( the synaptic cleft ) to receptors on the surface of a recipient , or postsynaptic , neuron
神经元放电时，电性讯号沿著轴突传至神经末梢（突触前终端） ，造成神经传递物从囊泡释放到细胞外，并通过微小的细胞间空隙（突触间隙） ，抵达接收端，也就是突触后神经元表面的受体。 |
