Dr casanova thinks hse is the first example of a disease that was thought to be purely infectious but which has turned out to be purely monogenic ? that is , under the control of a single gene 与适应性免疫相反,先天免疫与生俱来的,而非后天由于人们接触致病菌而形成抗体所获得的。
2.
All f1 seedlings showed wild - type salt - sensibility , and two of four mutants were detected that the proportion of the salt - tolerance seedlings to salt - sensibility seedlings was 1 : 3 in f2 progenys . the results of genetic analysis suggested that this mutants have a monogenic recessive mutation in a nuclear gene F _ 1代白花授粉所得的f _ 2代中,两个株系抗盐植株与不抗盐植株的比例为1 : 3 ,表明该突变体是隐性单基因突变体。
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In this paper , resorting to the hypermonogenic function in real clifford analysis , we define the hypermonogenic function in complex clifford analysis and give the sufficient and necessary conditions of complex monogenic and complex hypermonogenic functions . the result is similar to the cauchy - riemann condition of complex analysis . so we get some relations between the real and complex clifford functions 本文借助实clifford分析中的超正则函数,定义了复clifford分析中的超正则函数,得到了复正则函数及复超正则函数的充分必要条件,这些条件类似于单复变中的cauchy - riemann条件,使复clifford函数与实clifford函数有了联系,并讨论了复超正则函数的若干性质。
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We obtain two recessive monogenic salt - tolerance mutants from co60 - - mutagenized arabidopsis thaliana m2 populations . the effect of nacl on the structure of vegetative organa in arabidopsis thaliana was further studied and through a rapd analysis on salt - tolerance mutants of arabidopsis thaliana , a 1200bp dna fragment probably related to the salt - tolerance gene was get 本实验以模式植物拟南芥( arabidopsisthaliana )为材料获得了隐性单基因抗盐突变体,并研究了不同浓度的nacl胁迫对拟南芥营养器官结构的影响,进而通过对突变体的rapd分析,获得了一个与植物抗盐性有关的1200bp大小的基因片段。
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This article reviews the clinical manifestations , mutation feature , gene location and phenotype of different ischemic cerebrovascular disease caused by monogenic disorders , including coagulation disorders , erythrocytic disorders , inherited small vessel disease , metabolic disorders , connective tissue diseases , vasculopathies and disorders of unknown etiology 本文主要阐述了单基因遗传障碍引起的缺血性脑血管病,包括凝血障碍、血细胞病、遗传性小血管病、代谢障碍、结缔组织病、大动脉病及不明原因引起缺血性脑血管病的临床特征、突变特点、基因定位及表型等遗传学研究进展。