| 1. | Methods : sepsis was induced by cecal ligation and puncture operation
方法；结扎和刺破盲肠造成脓毒血症。 |
| 2. | The research of hepatic configuration and function in septicemic rat
脓毒血症过程肝脏结构和功能关系的研究 |
| 3. | Herbal medicine for activating blood and transforming stasis on sepsis : a clinical study
活血化瘀法治疗脓毒血症42例疗效观察 |
| 4. | Background : sepsis attenuates the muscle - relaxing effects of nondepolarizing neuromuscular blockers
背景：脓毒血症能减弱非去极化神经肌肉阻滞剂的肌松作用。 |
| 5. | It is thought that these elicit the sepsis - induced attenuation of the muscle - relaxing effects of rocuronium
从中得出脓毒血症引起的减弱肌松剂罗库溴铵的作用。 |
| 6. | Endplate potentials , acetylcholine potentials , and electrotonic potentials were recorded from the motor endplates of isolated diaphragms from acute late septic and nonseptic rats
记录急性迟发性脓毒血症和非脓毒血症大鼠的离体横膈运动终板上的终板电位、乙酰胆碱电位和电紧张电位。 |
| 7. | Conclusions : these results indicate that acute late sepsis facilitates endplate potentials and enhances excitability of the muscle membrane , indicated by a decrease of critical depolarization
结论：这些结果显示急性迟发性脓毒血症易化终板电位，增强肌肉细胞膜的兴奋性，减少临界去极化。 |
| 8. | The authors investigated the effects of acute late sepsis on neuromuscular transmission and neuromuscular actions of rocuronium to clarify the mechanisms by which sepsis attenuates the effects of nondepolarizing neuromuscular blockers
笔者研究了急性迟发性脓毒血症在罗库溴铵的神经肌肉传递和作用上的效应来阐明脓毒血症减弱去极化神经肌肉阻滞剂作用的机理。 |
