| 1. | Hyperpolarization and repolarization both describe increased electronegativity within the cell .
超极化及再极化都表明细胞内负电性的增强。 |
| 2. | The hyperpolarization of membrane potential is referred to as the inhibitory junction potential ( ijp )
这种超极化被称为抑制性接点电位( inhibitoryjunctionpotential ， ijp ) 。 |
| 3. | Application of tea could block the outward current , and decrease the mean amplitudes of the initial and second phases of ach - induced hyperpolarization
实验结果提示ach对与ec共培养的smc影响可能与ec膜上的kc 。 |
| 4. | Previous evidence showed that an increase in [ ca2 + ] cyt has been shown to be an early event in the aba signal transduction pathway , and ca2 + channels at the plasma membrane of stomatal guard cells are activated by hyperpolarization and aba
Assmann已提出较为完善的气孔保卫细胞信号转导模式，但是对于aba信号转导的一些中间环节仍是人们不断探讨的问题。 |
| 5. | In addition , in the later of 80 ' s , the results obtained in weston and suzuki ' s laboratories and other laboratories showed that a new substance , christened edhf , could be released by ach or other vasoactive substances such as substance p . and the edhf could cause hyperpolarization of the vascular smooth muscle cells in an endothelium - dependent manner
除edrf外， 80年代末weston和suzuki等发现ach等血管活性物质还可使血管内皮释放一种可使smc膜电位超极化的因子( edhf ) 。 edhf介导的血管舒张不受血红蛋白和亚甲蓝的影响，说明其作用机制与no无关。 |
| 6. | The objectives of this study were : ( 1 ) to establish a model of co - culture of endothelial and smooth muscle cells ; ( 2 ) - 5 to ascertain the effects of the ach on the membrane potential of the rabbit aortic smooth muscle cells and bovine aortic endothelial cells ; ( 3 ) to clarify the role of gap junctions in ach - induced endothelium - derived hyperpolarization in rabbit aortic smooth muscle cells ; ( 4 ) to examine the contribution of both no and gap junction to the ach - induced endothelium - derived hyperpolarization in rabbit aortic smooth muscle cells
Edhf的性质尚未确定，有的研究认为其与ec的k ~ +通道有关，而有的研究认为平滑肌的内皮源性超极化与ec和smc之间的缝隙连接( gapjunction )有关，因为两者通过缝隙连接有着电偶联作用。本文利用细胞共培养和膜片钳技术，探讨缝隙连接在血管张力调节中的作用。 |
