肺泡上皮细胞 meaning in Chinese
alveolar epithelial cells
pulmonary epithelial cell
Examples
- Downregulation of gap junctional intercellular communication induced by silicon dioxide in the pulmonary alveolar epithelial cen
2刺激肺泡上皮细胞间隙连接功能下调的研究 - Scientists at the imm used the in itro method to create lung epithelial cells known as aleolar epithelial type ii
Imm的科学家以这种方法在体外制出了肺上皮细胞?又名ii型肺泡上皮细胞。 - It was firstly observed that cgrp plays a protective role on lung injury and attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis . the protective mechanisms of cgrp are not mediated by blocking no production
并进一步观察到rp具有抗iu所致肺泡上皮细胞损伤的保护作用，可减轻o对ps脂质合成的抑制，但cgrp的作用并不是通过阻断o激活nos ，使no生成减少而实现的。 - Conclusions electron microscopic obser vations show that the alveolar epithelial cells , endothelial cells and basement membrane are all injured , interstitial macrophages hyperpplasia , mast cells infiltration and degranulation , electron - dense deposits at alveolar wall are present
结论电镜观察可见特发性肺纤维化时肺泡上皮细胞、血管内皮细胞及基底膜均有损伤，并可见间质巨噬细胞增生，肥大细胞浸润及脱颗粒，肺泡壁电子致密物沉积。 - Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants , nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups , mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801 , l - nna could block the release of ldh induced by glu
谷氨酸对肺表面活性物质脂质合成抑制的机制研究nmda受体拮抗剂mk - 801能逆转glu所致肺组织[ ~ 3h ] -胆碱掺入减少的效应；肺泡上皮细胞有nmda受体nmdar1亚单位的表达； glu对ps脂质合成的影响依赖l -精氨酸的存在， glu可使肺组织匀浆中nos活性、 no合成量升高； mk - 801 、 nos的抑制剂l - nna均可阻断glu所致的这些效应； l - nna可逆转glu所致肺组织[ ~ 3h ] -胆碱掺入减少的效应； glu可使肺组织匀浆中mda硕士学位论文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻断q所致的这些效应； mk80 、 l nna还能阻断gill诱导的ldh释放增多的效应。